More are losing jobs because ICE has deleted their visas or names from the SEVIS federal database that allows migrants to get work permits after enrolling in U.S. colleges even with prior criminal activity — such as shoplifting, drunk driving, or street altercations that Biden Admin allowed
Hundreds of Indian and Chinese college-graduate migrants in the United States have been fired from white-collar jobs in California, Washington, DC, and elsewhere,
But the migrants are pushing back with lawyers, complaints in the Indian media, and pleas to Indian-origin members of Congress.
Some are losing their jobs at Apple and the Federal National Mortgage Association, commonly known as Fannie Mae.
More are losing jobs because the Immigration and Customs Enforcement agency has deleted their visas or their names from a federal database — dubbed SEVIS — that allows migrants to get work permits after enrolling in U.S. colleges. Their entries and work permits are being deleted from SEVIS because of their prior criminal activity — such as shoplifting, drunk driving, or street altercations — that was ignored by President Joe Biden’s pro-migration deputies.
In March, Citigroup announced it would sharply reduce its reliance on foreign contractors after the contractors cost the bank at least $600 million in routine operations and regulatory incompetence. The decision means Citibank will begin hiring at least 10,000 American technology professionals for jobs held by supposedly cheaper Indian workers.
All this is good news for the many American professionals who have lost jobs to the federal government’s mass inflow of mixed-skill Indian graduates. Most of the Indian migrants use their H-1B visas or university-related work permits to take roughly 1 million jobs that would otherwise go to skilled American professionals. Many of the migrants are hired for Fortune 500 jobs by ethnic networks that discriminate against Americans.
Many American white-collar workers hope President Donald Trump’s deputies will recognize the huge damage done by legalized white-collar migration — partly because Trump launched major reforms in 2020. His reforms were quickly wrecked by President Joe Biden’s pro-migration deputies.
“There are reasons to be optimistic about the administration … but it’s going to take time,” Kevin Lynn, founder of US Tech Workers, told Breitbart News. “This time to be very aggressive” to demand that politicians constrict the multiple visa-worker programs that slash Americans’ salaries, shrink professional power against teh C-Suite, and stall innovation, Lynn said.
But ethnic Indian politicians and ethnic associations are already pushing for the federal government jobs to be given back to the fired Indian visa workers.
“It has been brought to my attention that Fannie Mae has accused hundreds of my constituents in the Indian-American community of fraudulent behavior and fired them without conducting a full investigation or providing evidence,” said an April 9 letter from Rep. Suhas Subramanyam (D-VA), Rep. Raja Krishnamoorthi (D-IL), and Indian-born Rep. Shri Thanedar (D-MI). “We also request a briefing for ourselves and our staff at your earliest convenience,” the letter said.
Subramanyam’s district voted for Kamala Harris over Trump by 53 percent to 44 percent.
Fannie Mae has released little information about the nature and scale of the alleged fraud. “We fired over 100 employees from Fannie Mae who we caught engaging in unethical conduct, including facilitating fraud, against our great company,” said an April 4 statement from William Pulte, Chairman of the Board of Directors of Fannie Mae.
The scandal has been closely followed by the Indian press, which notes that most of the Indians in the Fannie Mae firing were from the ethnic Telugus from the distinct province of Telangana.
The Times of India reported on April 16:
Some employees are alleged to have colluded with non-profit organizations like the Telugu Association of North America (TANA) and misused company funds. Incidentally, this is the same scam that saw the Cupertino-based company’s firing some Indian-origin employees earlier this year. One of the employees who was laid off at Fannie Mae reportedly held the position of regional vice president in TANA. Sources told Time that TANA is not alone in the scam, and other organizations are also likely involved.
In the Apple firings, India Today described the alleged fraud in a January 8 article about the mass firings:
The reported scheme revolves around Apple’s Matching Grants program, a corporate social responsibility initiative designed to amplify employees’ charitable contributions by matching their donations to nonprofit organisations. The allegations suggest that some employees, in collaboration with specific nonprofit organisations — including associations reportedly linked to the Indian community — falsified donations to exploit the program.
The District Attorney’s office in Santa Clara described the charges against several Chinese-origin people who were hired for jobs at Apple:
Over three years, the six tech employees defrauded both the State of California and Apple’s Matching Gifts Program. Under the direction of Siu Kei (Alex) Kwan, five defendants all pretended to make donations to the American Chinese International Cultural Exchange (ACICE) or Hop4Kids. In fact, they were given the donations back and Kwan – the CEO of Hop4Kids and accountant for ACICE – kept Apple’s matching funds. Meanwhile, all the donators wrote off their “charitable donations” on their tax returns.
….
By exploiting Apple’s employee gift-matching program, the defendants extracted approximately $152,000 from Apple’s program and overreported around $100,000 in charitable contributions as tax deductions.
Many Indians are losing jobs because the Department of Homeland Security is removing migrants’ names from the SEVIS database needed for work permits. The revocations have mostly been stalled by more than 28 lawsuits on behalf of more than 1,500 migrants from many countries.
This population includes Indians who got work permits via the Curricular Practical Training (CPT) program or the Occupational Practical Training (OPT) program. In 2023, almost 450,000 migrants held work permits after paying tuition to a wide variety of colleges. The colleges range from low-quality colleges with minimal coursework to better-quality classes in elite Ivy League universities.
“These sudden revocations have left many students in shock, facing the grim reality of deportation and the invalidation of their legal stay, Form I-20, and Employment Authorization Documents (EAD),” said GG2.net, a news site based in Gujarat province. The article continued:
Among the impacted students is a Hyderabad native currently studying in Omaha, who revealed he was fined two years ago for speeding in New York but was never arrested. Another student admitted to a DUI (driving under the influence) charge, saying he had followed all court mandates, including installing a car ignition lock and undergoing sobriety checks.
In a more severe case, a Texas-based student was arrested for shoplifting $144 worth of items from Walmart. However, the case was dismissed due to his clean academic record and cooperation. “I followed all legal instructions and thought the matter was behind me. Why am I being punished again?” he asked.
The administration is defending its actions amid a flurry of lawsuits from students, employer groups, and pro-migration groups.
Border agents may also be enforcing anti-fraud rules to block the airport arrival of white-collar migrants, many of whom face huge hometown pressure to find jobs in the United States
There is also some initial evidence that President Donald Trump’s deputies want to do something about routine anti-American discrimination by Indian companies in the United States. According to Bloomberg:
The US Equal Employment Opportunity Commission is investigating dozens of American workers’ allegations that India’s biggest IT outsourcer, Tata Consultancy Services Ltd., discriminated against them based on their race, age and national origin.
The former employees are largely professionals from non-South Asian ethnic backgrounds over the age of 40, who say the company targeted them for layoffs but spared Indian colleagues, some of whom were working on H-1B skilled worker visas. They began filing complaints against TCS in late 2023.
…
The EEOC is tasked with enforcing laws prohibiting discrimination in the workplace. In 2020, its investigation of another of the world’s biggest outsourcing firms, Cognizant Technology Solutions Corp., found that the company discriminated against non-Indian workers in its US operation. A jury in a federal class-action lawsuit found in October that Cognizant intentionally discriminated against more than 2,000 non-Indian employees between 2013 and 2022, echoing the EEOC findings. Cognizant said it provides equal employment opportunities for all employees and does not tolerate discrimination. It has said it plans to appeal the verdict and disagrees with the EEOC finding.
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REDEMPTION FOR THE VACCINE-PURGED: Secretary Pete Hegseth Issues Powerful Reinstatement Order for Troops Removed by ‘Unlawful Experimental COVID-19 Jab Mandate’
Defense Secretary Pete Hegseth announced sweeping reforms to address the catastrophic fallout from the Biden administration’s unlawful COVID-19 vaccine mandate.
Speaking with the conviction of a leader determined to rebuild trust, Hegseth outlined the Department of Defense’s (DOD) aggressive efforts to reinstate service members and veterans who were unfairly punished for standing by their principles.
The Gateway Pundit previously reported on military veterans’ concerns about a new Department of Defense reinstatement form, which they claim hides the truth about their forced separations during the COVID-19 vaccine mandate era.
They are being told that if they want back in, they must lie on paper and pretend they left the military of their own free will.
According to the form: “I, [name], attest I voluntarily separated from the (Air) (Space) Force or allowed my service to lapse rather than be vaccinated under the COVID-19 vaccine mandate, which was in effect from 24 August 2021 to 10 January 2023. My decision to separate was made freely and without coercion.”
The DoD’s response team immediately issued a statement, stating they’re “reviewing” the matter.
In a video statement posted Wednesday, Hegseth offered an apology “for how it was done, and we want to fix it.”
Effective immediately, the Department of Defense is scrubbing the records of all troops who were punished for refusing the experimental jab—many of whom were discharged with less-than-honorable designations that tarnished their careers and cut off benefits.
Constitutional attorney and retired LTC Green Beret Ivan Raiklin issued a powerful statement following this announcement from Hegseth.
“Did you just say “illegal DOD jab mandate”? Boom. You caught my attention, sir. I’ve just upgraded your grade as Secretary of Defense to an A. You want to achieve an A-plus? Bring me in—recall me to active duty—and I will gladly, pro bono even, come in and lead the Nuremberg 2.0 trials,” said Raiklin.
“The first defendants that need to be addressed are Lloyd Austin and Mark Milley. Why? Because they conducted mutiny by removing—kicking out, like you said—8,700. And then the 10, almost 100,000 of us that left because we were not going to comply with the DOD jab mandate.”
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Childhood Exposure to Bacterial Toxin May Be Triggering Colorectal Cancer Epidemic Among the Young
n an effort to explain a modern medical mystery, an international team of researchers led by the University of California San Diego has identified a potential microbial culprit behind the alarming rise in early-onset colorectal cancer: a bacterial toxin called colibactin.
Produced by certain strains of Escherichia coli that reside in the colon and rectum, colibactin is a toxin capable of altering DNA. Now, scientists report that exposure to colibactin in early childhood imprints a distinct genetic signature on the DNA of colon cells—one that may increase the risk of developing colorectal cancer before the age of 50.
The new study, published on April 23 in Nature, analyzed 981 colorectal cancer genomes from patients with both early- and late-onset disease across 11 countries with varying colorectal cancer risk levels. The findings show that colibactin leaves behind specific patterns of DNA mutations that were 3.3 times more common in early-onset cases (specifically in adults under 40) than in those diagnosed after the age of 70. These mutation patterns were also particularly prevalent in countries with high incidence of early-onset cases.
“These mutation patterns are a kind of historical record in the genome, and they point to early-life exposure to colibactin as a driving force behind early-onset disease,” said study senior author Ludmil Alexandrov, professor in the Shu Chien-Gene Lay Department of Bioengineering and the Department of Cellular and Molecular Medicine at UC San Diego, who is also a member of UC San Diego Moores Cancer Center and Deputy Director of Sanford Stem Cell Fitness and Space Medicine Center.
Although previous studies—including earlier work from Alexandrov’s lab— have identified colibactin-related mutations in roughly 10 to 15 percent of all colorectal cancer cases, those studies either focused on late-onset cases or did not distinguish between early- and late-onset disease. This latest study is the first to demonstrate a substantial enrichment of colibactin-related mutations specifically in early-onset cases.
The implications are sobering. Once considered a disease of older adults, colorectal cancer is now on the rise among young people in at least 27 countries. Its incidence in adults under 50 has roughly doubled every decade for the past 20 years. If current trends continue, colorectal cancer is projected to become the leading cause of cancer-related death among young adults by 2030.
Until now, the reasons behind this surge have remained unknown. Young adults diagnosed with colorectal cancer often have no family history of the disease and few known risk factors such as obesity or hypertension. That has fueled speculation about potential hidden environmental or microbial exposures—something this new study directly investigates.
“When we started this project, we weren’t planning to focus on early-onset colorectal cancer,” said study co-first author Marcos Díaz-Gay, a former postdoctoral researcher in Alexandrov’s lab. “Our original goal was to examine global patterns of colorectal cancer to understand why some countries have much higher rates than others. But as we dug into the data, one of the most interesting and striking findings was how frequently colibactin-related mutations appeared in the early-onset cases.”
According to the team’s analysis, colibactin’s damaging effects begin early. By molecularly timing each mutational signature identified in this study, the researchers demonstrate that colibactin-associated mutations arise early in tumor development, consistent with prior studies showing that such mutations occur within the first 10 years of life. The study also reveals that colibactin-related mutations account for approximately 15% of what are known as APC driver mutations—some of the earliest genetic alterations that directly promote cancer development—in colorectal cancer.
“If someone acquires one of these driver mutations by the time they’re 10 years old,” Alexandrov explained, “they could be decades ahead of schedule for developing colorectal cancer, getting it at age 40 instead of 60.”
In other words, colibactin-producing bacteria may be silently colonizing children’s colons, initiating molecular changes in their DNA, and potentially setting the stage for colorectal cancer long before any symptoms arise.
Alexandrov cautioned that while their findings provide strong support for this hypothesis, further research is necessary to establish causality.
Building on past breakthroughs
This work—part of Cancer Grand Challenges team Mutographs, funded by Cancer Research UK—is the latest milestone in a growing body of research that Alexandrov, Díaz-Gay and colleagues have been advancing over the past several years. Their specialty lies in decoding patterns of DNA mutations caused by environmental exposures—such as UV radiation and bacterial toxins—and lifestyle behaviors like smoking and drinking. Each factor leaves a distinct genetic fingerprint in the genome, a unique mutational signature that can help pinpoint the origins of certain cancers.
As part of a long-term collaboration between UC San Diego, the International Agency for Research on Cancer (France), and the Wellcome Sanger Institute (UK), enabled by Cancer Grand Challenges funding, team Mutographs has elucidated the mutational processes underlying esophageal, kidney, and head and neck cancers worldwide. This most recent result on colorectal cancer further expands the global understanding of cancer etiology through mutational signature analysis.
By systematically cataloging these mutational patterns across thousands of cancer genomes, the researchers have been working to identify new causes of cancer that had previously flown under the radar.
“Not every environmental factor or behavior we study leaves a mark on our genome,” said Alexandrov. “But we’ve found that colibactin is one of those that can. In this case, its genetic imprint appears to be strongly associated with colorectal cancers in young adults.
The team’s approach—using genomic detective work to uncover hidden causes of cancer—demonstrates the power of fundamental scientific research. But unfortunately, the future of this work is in peril.
Although the research has been supported in large part by programs in the UK such as Cancer Research UK via Cancer Grand Challenges, a substantial portion of the team’s funding has come from the U.S. National Institutes of Health (NIH). With the NIH now facing proposed budget cuts, critical projects like this one could be at risk.
“If NIH funding cuts impact our ability to do this work, that will be, in my opinion, a substantial hit to cancer research not just in the U.S., but globally,” said Alexandrov. “Our funding has allowed us to collaborate with cancer researchers around the world, collecting and analyzing large datasets from patient samples in multiple countries. That kind of scale is what makes discoveries like this possible.”
Ongoing work
Continued support is especially important for the next phase of their research, as it raises a host of new questions. How are children being exposed to colibactin-producing bacteria, and what can be done to prevent or mitigate that exposure? Are certain environments, diets or lifestyle behaviors more conducive to colibactin production? How can people find out if they already have these mutations?
The team is investigating several hypotheses while also further examining the correlation between colibactin and the risk of early-onset colorectal cancer. In addition, they are exploring whether the use of probiotics could safely eliminate harmful bacterial strains. They are also developing early detection tests that analyze stool samples for colibactin-related mutations.
But these efforts will require resources.
“To further investigate our hypotheses and develop safe, ethical interventions, we’re going to need tens of millions of dollars,” said Alexandrov. “This research has important implications for the future health of children globally. Without adequate support, it will be difficult to fully understand and address the issue.”
In the meantime, the team is continuing its global search for cancer-linked mutational signatures. In the recent Nature study, the team also found that colorectal cancers from specific countries—particularly Argentina, Brazil, Colombia, Russia and Thailand—showed an increase in certain mutational signatures. This suggests that local environmental exposures may also contribute to cancer risk.
“It’s possible that different countries have different unknown causes,” said Díaz-Gay, who is launching a new phase of the study in his newly established lab at the Spanish National Cancer Research Center (CNIO) in Madrid, Spain. “That could open up the potential for targeted, region-specific prevention strategies.”
Alexandrov noted a broader implication of the research: that many cancers may originate from environmental or microbial exposures in early life, long before diagnosis.
“This reshapes how we think about cancer,” he said. “It might not be just about what happens in adulthood—cancer could potentially be influenced by events in early life, perhaps even the first few years. Sustained investment in this type of research will be critical in the global effort to prevent and treat cancer before it’s too late.”
Full study: “Geographic and age variations in mutational processes in colorectal cancer.”
This work is supported in part by Cancer Research UK, the U.S. National Institutes of Health, David and Lucile Packard Foundation, UC San Diego Sanford Stem Cell Institute and the Wellcome Trust.
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